When Bacillus thuringiensis sporulates, it synthesizes protein crystals called delta-endotoxins, to which it owes its insecticidal activity. These protoxins need to be ingested by the larvae in order to act, since the selective toxicity of Bt for the larvae of certain insects is due to two factors in its mode of action:

$ Toxins require an alkaline medium for their activation, a characteristic that only occurs in the intestine of most insects.

$ Toxins can only act if they are bound to specific receptors, and this specificity depends on the insect.

Thus, each insect species, depending on the nature of its receptors, will be sensitive or not. When both factors are combined, the toxins quickly attach to their receptors and cause intestinal paralysis, preventing peristaltic movements, so the insect stops feeding. In addition, the intestinal epithelium breaks down, allowing intestinal fluids to pass to the rest of the insect's vital organs and tissues. Only a few hours after having ingested the spore with the toxin, the insect's mandibles become paralyzed and feeding stops. Later, the paralysis becomes generalized, reflex movements disappear and the larva dies when its heartbeat stops.

INDICATIONS:

It stands out for its action on agricultural pests: Heliothis, Pieris, Plusia, Plutella, Ostrinia, Capua, Prays and Cacoecia and forest pests: Lymantrinia, Malacosoma, Euproctis and Tortryx viridiana. DOSE: 0.5-1.5 Kg/ha

APPLICATION:

It can be applied using conventional spraying equipment, wetting the entire plant well and in the early stages of the larva.

INCOMPATIBILITIES:

With anilazine, captafol, captan, demeton S-ethyl, difolatan, dimethoate, dinocap, phenthoate, fosalon, isoprocarb, leptophos, propargite, propoxur and tetrachlorvinphos; There should be three days between treatments with these products and with B. thuringiensis and vice versa.